Cat. No. | Product Name | Species | Expression System |
---|---|---|---|
TMPY-05094 |
Tissue Factor Protein, Cynomolgus, Recombinant (His)
coagulation factor III (thromboplastin, tissue<... |
Cynomolgus | HEK293 Cells |
Tissue Factor Protein, Cynomolgus, Recombinant (His) is expressed in HEK293 mammalian cells with His tag. The predicted molecular weight is 26.4 kDa and the accession number is XP_005542723.1. | |||
TMPK-00525 |
KLKB1 Protein, Cynomolgus, Recombinant (His)
KLK3,Plasma kallikrein,KLKB1,PKK,Kininogenin,Fletcher fa... |
Cynomolgus | HEK293 Cells |
Plasma kallikrein, also known as Fletcher factor or kallikrein B1 (KLKB1), is a serine endopeptidase, like its homologs tissue kallikrein and kallikrein-related peptidases (KLKs). Its physiological role is to catalyze the release of kinins and other vasoactive peptides. KLKB1 Protein, Cynomolgus, Recombinant (His) is expressed in HEK293 mammalian cells with C-His tag. The predicted molecular weight is 70.19 kDa and the accession number is A0A2K5VTJ9. | |||
TMPK-00532 |
CD46 Protein, Cynomolgus, Recombinant (His)
CD46,MCP,TLX,MIC10,AHUS2,TRA2.10 |
Cynomolgus | HEK293 Cells |
CD46 was discovered in 1986 during a search for novel C3b-binding proteins. CD46 is expressed ubiquitously and functions as a co-factor in the factor I-mediated proteolytic cleavage of C3b and C4b. Its vital role in preventing complement deposition on host tissue is underpinned by the fact that deficiency of CD46 is a predisposing factor for numerous disease conditions arising from complement-mediated 'self-attack'. CD46 Protein, Cynomolgus, Recombinant (His) is expressed in HEK293 mammalian cel... | |||
TMPK-00512 |
IL-18R alpha Protein, Cynomolgus, Recombinant (aa 22-329, His)
IL18R1,IL-1 Rrp,CD218a,IL18Ra,IL-18Ra,IL-18 R alpha,IL1RRP,I... |
Cynomolgus | HEK293 Cells |
Interleukin-18 (IL-18) is a member of the interleukin-1 family of cytokines produced constitutively by different cell types and by adipose tissue. Interleukin (IL)-18 was originally discovered as a factor that enhanced IFN-γ production from anti-CD3-stimulated Th1 cells, especially in the presence of IL-12. Upon stimulation with Ag plus IL-12, naïve T cells develop into IL-18 receptor (IL-18R) expressing Th1 cells, which increase IFN-γ production in response to IL-18 stimulation. IL-18R alpha Pr... |